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Tumor-derived prostaglandin E2 (PGE2) restricts the proliferative expansion and effector differentiation of TCF1+ stem-like CD8+ T cells within tumors, promoting cancer immune escape.


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Tumour-derived prostaglandin E2, signaling through its receptors EP2 and EP4, is shown to restrain the responses of tumour-infiltrating stem-like TCF1+CD8+ T lymphocytes, and modulation of&nbsp;T cell EP2 and EP4 can restore anticancer immunity.

PGE2 limits effector expansion of tumour-infiltrating stem-like CD8+ T cells - Nature

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Prostaglandin E2 Suppresses Expansion and Effector Differentiation of Tumor-Infiltrating Stem-like CD8+ T Cells


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Tumor-infiltrating stem-like CD8+ T cells and the immunosuppressive role of prostaglandin E2